Lung Cancer Risk Factors

Lung Cancer Risk Factors

lung-cancer-risk-factors-exposure

Cancer is a genetic disease and is the result of abnormal changes in the DNA during cell division. These changes are called mutations. Mutations happen in two different ways, either through somatic  (cells of our body) or germline mutation (mutations passed on from parents to offspring). The first type of gene alteration is the result of the accumulation of harmful changes in the cells of the body (somatic) during division. As you see, the somatic mutation is not inherited but is the result of a detrimental error in the replication of the DNA every time the somatic cell divides. The errors’ accumulation is due to a myriad of environmental carcinogens, age, and lifestyle. This includes smoking, alcohol, ionizing exposure, and chemical exposure. The second type of mutation is passed from parents to their offspring. Hyperplasia is the first step for a tumor to develop. It is a rapid increase in the cells` number. Although, it does not always lead to cancer. Examples for a physiological rapid cell division are an embryo when developing in the womb or when a wound is healing. The second step of precancerous formation is dysplasia. During this stage, cells do not stop to divide, and the more the division continues. As a result, they lose their differentiation or resemblance to a normal cell. This process leads to a dysfunctional cell. Researchers consider hyperplasia and dysplasia a precancerous formation but it could take ages to become cancer.​  

 

Lung Cancer Risk Factors / Chemical Exposure

Lung Cancer Risk Factors / Genetic Heritage

Lung Cancer Risk Factors / Personal Behavior

Other Risk Factors